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Employees celebrating STAR Awards.

STAR Awards recognize dedicated UTHealth Houston employees

The UTHealth Houston STAR Awards reception and luncheon celebrated employees and recognized milestone years of service ranging from five to 45 years.
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Photo of the UCT building.

UT System Board of Regents to launch search for new president of UTHealth Houston

University of Texas System Board of Regents Chairman Kevin P. Eltife has appointed a presidential search advisory committee to support the regents and chancellor on selecting the next president of UTHealth Houston.
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Five UTHealth Houston employees standing behind a table with a laptop on it

UTHealth Houston earns five 2024 Top Workplaces National Culture Excellence Awards

UTHealth Houston has been honored with five prestigious 2024 Top Workplaces National Culture Excellence Awards, marking a significant achievement in workplace culture and employee satisfaction.
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J. William Lindsey, MD, senior author of the study and professor in the Department of Neurology with McGovern Medical School at UTHealth Houston. (Photo by UTHealth Houston)

UTHealth Houston study: EBV-specific T-cells play key role in development of multiple sclerosis

The body’s immune response to Epstein-Barr virus (EBV) may play a role in causing damage in people with multiple sclerosis, according to a new study led by UTHealth Houston.
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Paul E. Schulz, MD, the Rick McCord Professor in Neurology with McGovern Medical School at UTHealth Houston, was senior author of a study that found several vaccinations were linked to a reduced risk of Alzheimer's disease. (Photo by UTHealth Houston)

Several vaccines associated with reduced risk of Alzheimer’s disease in adults 65 and older

Prior vaccination against tetanus and diphtheria, with or without pertussis (Tdap/Td); herpes zoster (HZ), better known as shingles; and pneumococcus are all associated with a reduced risk for developing Alzheimer’s disease, according to new research from UTHealth Houston.
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Photo of Diana Milewicz, PhD.

Researchers uncover how a genetic mutation can cause individuals with normal cholesterol levels to develop coronary artery disease at a young age

A novel molecular pathway to explain how a mutation in the gene ACTA2 can cause individuals in their 30s – with normal cholesterol levels and no other risk factors — to develop coronary artery disease has been identified, according to researchers with UTHealth Houston.
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